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Please use this identifier to cite or link to this item: https://hdl.handle.net/1/1668
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dc.contributor.authorRoger, Simon D-
dc.contributor.otherTee, S.L.-
dc.contributor.otherSindone, Andrew-
dc.contributor.otherAtherton, J.-
dc.contributor.otherAmerena, J.-
dc.contributor.otherD'Emden, M.-
dc.contributor.otherErlich, J.-
dc.contributor.otherDePasquale, C.-
dc.date.accessioned2019-09-30T06:35:57Z-
dc.date.available2019-09-30T06:35:57Z-
dc.date.issued2019-08-
dc.identifier.citation50(6):659-666en
dc.identifier.issn1444-0903en
dc.identifier.urihttps://elibrary.cclhd.health.nsw.gov.au/cclhdjspui/handle/1/1668-
dc.description.abstractHyponatraemia is common in heart failure (HF). It is estimated that over 20% of patients admitted to hospital with HF have hyponatraemia (1). It has also been repeatedly shown to be a surrogate marker of increased morbidity and mortality in this specific population (2). This review focuses on the pathophysiology of hyponatraemia through the activation of neurohormonal cascades in HF, the clinical implications of sustained hyponatraemia, as well as treatment options in the management of this challenging phenomenon. This article is protected by copyright. All rights reserved.en
dc.description.sponsorshipRenalen
dc.subjectCardiologyen
dc.subjectHeart Diseaseen
dc.titleHyponatremia in Heart Failureen
dc.typeJournal Articleen
dc.identifier.doi10.1111/imj.14624en
dc.description.pubmedurihttps://www.ncbi.nlm.nih.gov/pubmed/31449715en
dc.description.affiliatesCentral Coast Local Health Districten
dc.description.affiliatesGosford Hospitalen
dc.identifier.journaltitleInternal Medicine Journalen
dc.originaltypeTexten
item.fulltextNo Fulltext-
item.grantfulltextnone-
item.openairetypeJournal Article-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.cerifentitytypePublications-
Appears in Collections:Cardiology
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